QuantiChrom Urea Assay Kit (100T) is an assay kit that provides a simple and direct way for accurate quantitative measurement of the molecule(s) that it is designed for. The user friendly protocol will guide you step by step through the automation-ready procedures that are need for performing the analysis. By simply measuring the changes in colour, absorbance and optical density with the help of a microplate reader you will obtain data that is in direct correlation with the concentration of the molecule(s) of interest. With our QuantiChrom Urea Assay Kit (100T) precision and cost & time efficiency are guaranteed.
Storage and handling
QuantiChrom Urea Assay Kit (100T) is shipped at ambient temperature. Upon receiving the kit, please, store all components at +4 degrees Celsius (39.2 degrees Fahrenheit). The shelf life of all QuantiChrom kits varies between 6 and 12 months (reffer to the particular datasheet for the exact period).
|Quantity / Size||100|
|Benefits||• Assay kit that are simple, convenient and cost effective • Superior in performance • Researchers need little-to-notime for assay optimization • Specialize for both routine laboratory tests and for high-throughput drug discovery applications • with a focus on safe, non-radioactive assay|
|Description||Quantitative determination of urea by chemical colorimetric (520nm) method. Procedure: 30 min. Detection limit: 0.08 mg/dL (13 M). Shelf life: 12 months. Shipping: ambient temp; storage: 4, -20C.
Please note: since Jan 1, 2016, DIUR-500 (500 assays) is discontinued. The standard kit is DIUR-100 (100 assays).
|Key Features||Key features of our assays include simplicity, high-throughput, sensitivity, accuracy and low interference.|
|Storage Temp||4, -20°C|
|Shipping Conditions||room temp|
|References||Ramalingam, TR et al (2008) – Unique functions of the type II interleukin 4 receptor identified in mice lacking the interleukin 13 receptor alpha1 chain. Nat Immunol 9(1):25-33.
Sekiya S, Suzuki A (2011) – Direct conversion of mouse fibroblasts to hepatocyte-like cells by defined factors. Nature 475(7356):390-3
Stanic, AK et al (2006) – Immune dysregulation accelerates atherosclerosis and modulates plaque composition in systemic lupus erythematosus. PNAS 103(18):7018-23.